|
adherens junction disassembly
|
GO_0120179 |
[The disaggregation of an adherens junction into its constituent components. An adherens junction is a cell-cell junction composed of the epithelial cadherin-catenin complex at which the cytoplasmic face of the plasma membrane is attached to actin filaments.] |
|
regulation of anterograde synaptic vesicle transport
|
GO_1903742 |
[Any process that modulates the frequency, rate or extent of anterograde synaptic vesicle transport.] |
|
regulation of synaptic vesicle transport
|
GO_1902803 |
[Any process that modulates the frequency, rate or extent of synaptic vesicle transport.] |
|
TCR signalosome
|
GO_0036398 |
[A multi-protein complex containing at least the T-cell receptor complex and the LAT (linker for activation of T cells) scaffold protein. Also contains a variety of signaling proteins including co-receptors, kinases, phosphatases and adaptors such as CD8. Connects events on the plasma membrane to distal signaling cascades to ultimately modulate T cell biology.] |
|
obsolete canonical Wnt signaling pathway involved in cardiac neural crest cell differentiation involved in heart development
|
GO_0061310 |
[OBSOLETE. The series of molecular signals initiated by binding of a Wnt protein to a frizzled family receptor on the surface of the target cell, followed by propagation of the signal via beta-catenin, and ending with a change in transcription of target genes involved in cardiac neural crest cell differentiation.] |
|
obsolete negative regulation of phosphatidate phosphatase activity
|
GO_1903741 |
[OBSOLETE. Any process that stops, prevents or reduces the frequency, rate or extent of phosphatidate phosphatase activity.] |
|
negative regulation of torso signaling pathway
|
GO_0120177 |
[Any process that stops, prevents, or reduces the frequency, rate or extent of the torso signaling pathway.] |
|
regulation of torso signaling pathway
|
GO_0120175 |
[Any process that modulates the frequency, rate or extent of the torso signaling pathway.] |
|
obsolete canonical Wnt signaling pathway involved in cardiac muscle cell fate commitment
|
GO_0061317 |
[OBSOLETE. The series of molecular signals initiated by binding of a Wnt protein to a frizzled family receptor on the surface of the target cell, followed by propagation of the signal via beta-catenin, and ending with a change in transcription of target genes. In this pathway, the activated receptor signals via downstream effectors that result in the inhibition of beta-catenin phosphorylation, thereby preventing degradation of beta-catenin and contributing to cardiac muscle cell fate commitment. Stabilized beta-catenin can then accumulate and travel to the nucleus to trigger changes in transcription of target genes.] |
|
positive regulation of anterograde synaptic vesicle transport
|
GO_1903744 |
[Any process that activates or increases the frequency, rate or extent of anterograde synaptic vesicle transport.] |
|
positive regulation of synaptic vesicle transport
|
GO_1902805 |
[Any process that activates or increases the frequency, rate or extent of synaptic vesicle transport.] |
|
positive regulation of torso signaling pathway
|
GO_0120176 |
[Any process that activates or increases the frequency, rate or extent of the torso signaling pathway.] |
|
canonical Wnt signaling pathway involved in heart development
|
GO_0061316 |
[The series of molecular signals initiated by binding of a Wnt protein to a frizzled family receptor on the surface of the target cell, followed by propagation of the signal via beta-catenin, and ending with a change in transcription of target genes that contributes to the progression of the heart over time. In this pathway, the activated receptor signals via downstream effectors that result in the inhibition of beta-catenin phosphorylation, thereby preventing degradation of beta-catenin. Stabilized beta-catenin can then accumulate and travel to the nucleus to trigger changes in transcription of target genes.] |
|
negative regulation of anterograde synaptic vesicle transport
|
GO_1903743 |
[Any process that stops, prevents or reduces the frequency, rate or extent of anterograde synaptic vesicle transport.] |
|
negative regulation of synaptic vesicle transport
|
GO_1902804 |
[Any process that stops, prevents or reduces the frequency, rate or extent of synaptic vesicle transport.] |
|
obsolete canonical Wnt signaling pathway involved in positive regulation of cardiac muscle cell proliferation
|
GO_0061315 |
[OBSOLETE. The canonical Wnt signaling pathway that contributes to an expansion of the population of cardiac muscle cells.] |
|
positive regulation of nematode pharyngeal pumping
|
GO_1903746 |
[Any process that activates or increases the frequency, rate or extent of nematode pharyngeal pumping.] |
|
regulation of nematode pharyngeal pumping
|
GO_0043051 |
[Any process that modulates the contraction and relaxation movements of the pharyngeal muscle that mediates feeding in nematodes.] |
|
stress-induced homeostatically regulated protein degradation pathway
|
GO_0120174 |
[A stress-inducible protein catabolic pathway that promotes protein quality control by accelerating the degradation of misfolded ER membrane and cytosolic proteins, as well as native proteins. The pathway starts with the activation, by stress, of the Nma111p/Ynm3p serine protease, which cleaves the stress-induced hydrophilin Roq1p, resulting in the generation of a Roq1p cleavage product that selectively interacts with Ubr1p, an E3 ubiquitin ligase. Interaction with the Ubr1p type-1 substrate binding site reprograms the substrate specificity of this ubiquitin ligase resulting in the selective proteasome-mediated degradation of misfolded and native proteins. The pathway ends with degradation of the protein by the cytoplasmic proteasome. Currently, NMA111, ROQ1, UBR1, RAD6, and CDC48 are considered to be involved in this quality control pathway.] |
|
Notch signaling involved in heart development
|
GO_0061314 |
[The series of molecular signals initiated by binding of an extracellular ligand to a Notch receptor on the surface of the target cell and contributing to the progression of the heart over time.] |